Dr. Wernig's lab focuses on understanding the pathomechanisms of end stage organ fibrosis with the ultimate goal to help identify new targets for effective therapies. They found that the transcription factor c-JUN is specifically activated in human idiopathic pulmonary fibrosis and many other human fibrotic diseases and that induction of c-Jun in mice mimics the pathological hallmarks of these diseases.
James H. Clark Center, Stanford University 318 Campus Drive Stanford, CA 94305 Phone: 650.724.3333
Follow @StanfordBioX
© Stanford University, Stanford, California 94305
